This newsletter reveals how calcium dysregulation is not a byproduct of type 2 diabetes but a driving force behind β-cell failure, insulin resistance, and early vascular damage—redefining how metabolic dysfunction begins.
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Calcium isn’t just present in pain pathways—it’s actively shaping them at every level. Pain starts when calcium channels in nociceptors open, allowing an ion surge that triggers action potentials and sets pain in motion. At the spinal cord, this calcium influx causes sensory neurons to release pain transmitters like substance P and CGRP, pushing the signal one step further.
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Mental fatigue creeps in unnoticed, stealing focus, dulling motivation, and slowing the mind. But what if the key to restoring clarity and energy isn’t about pushing harder but about unlocking a fundamental cellular process?
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Part four explores the link between calcium imbalance and heart failure.
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Part three explores how calcium dysregulation contributes to hypertension and stroke risk. Part four explores the link between calcium imbalance and heart failure.
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Part two explores how anxiety disrupts heart function through calcium imbalance and why addressing it is key to cardiovascular health.
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Part one focuses on how ionic calcium regulates heartbeat rhythm, myocardial contraction, and overall cardiac performance.
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Ionic calcium plays a key role in regulating matrix metalloproteinases (MMPs)—enzymes that, when overactive, can break down the extracellular matrix in gingival tissues, leading to gum recession.
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